Vox Posted September 14, 2010 Report Posted September 14, 2010 Does anyone have a clue what this claimed new antioxidant would be and what would be its chemical structure?If it would be as efficient as claimed in the video it would need to effect directly to each cell, mitochondria and to the DNA , telomeres itself..?It should be both water and fat soluble to be universal antioxidant in the cell itself? Quote
HydrogenBond Posted September 18, 2010 Report Posted September 18, 2010 I do have not clue, but tend to think it will be more geared to reversing the signs of aging by tightening up wrinkles and fine lines. What is an interesting observation about aging are neurons stop reproducing quite early. These cells constantly rejuvenate themselves, through chemical exchange, and don't need to replicate for the rest of the life of the individual. If we assume the telomere theory is correct, neurons make the telomere last longer simply by not calling upon cell division, since they have the ability to keep what they have working in good order for decades. One anti-aging strategy is to make cells need to reproduce less, by teaching them the neuron secret of rejuvenation. One way to approach this is connected to another observation. Red blood cells can survive without nuclei or DNA. What that means is the cytoplasm or protein grid has some autonomy of its own, able to continue critical cells functions, without the DNA. It can't replicate but it can live on. The neuron sort of does this but with the DNA still there. The cytoplasm of cells has a level of functional autonomy, apart from the DNA, with the red blood cells showing this autonomy in plain form. This makes sense, since when we have cell cycles, the DNA has to be taken off-line to form chromosomes. So we need to depend on the autonomy of the protein grid to pick up the slack while DNA is asleep. With neurons, since the DNA does not replicate, one of the key drives of evolution has been taken away from neurons. In other words, not replicating takes away the traditional source of DNA mutation via replication. This limits changes to the neuron DNA using this basic evolutionary strategy If we have an organ with constant individual replication mutations in all its dividing cells, the needs of each become slightly different, such that there is more competition relative to the autonomous maintenance of each cell. But if they don't replicate it is easier to maintain group cooperation. Quote
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