Head Injury And Tumours

[paigetheoracle]

Is it true that no connection is seen between head injury and tumours as was mentioned on a BBC documentary in the UK?

 

To me it would seem that this is the body's way of dealing with scar tissue (broken blood vessels etc), by bundling it up in an easily managed area as after a traffic incident or a house fire, rescuing what we can from the disaster and clearing up the rest: Internal scab to grazed knee's equivalent. Could this also explain cancer and other tumours as internment camps for invaders or unwanted strays into forbidden areas?

[CraigD]

Is it true that no connection is seen between head injury and tumours as was mentioned on a BBC documentary in the UK?

I’ve never heard of a connection between head injury and tumors.

 

To me it would seem that this is the body's way of dealing with scar tissue (broken blood vessels etc), by bundling it up in an easily managed area as after a traffic incident or a house fire, rescuing what we can from the disaster and clearing up the rest: Internal scab to grazed knee's equivalent. Could this also explain cancer and other tumours as internment camps for invaders or unwanted strays into forbidden areas?

I don’t think so.

 

Scabs are just masses of special cells, mostly fibroblasts, that protect a wound from infection and help deliver other cells and raw protiens, especially collagen, to repair the wound. These cells are programmed for short lives – about 45 days or so – which is why scabs eventually crack and fall off.

 

Scar tissue are essentially collagen, long (as proteins go – about 0.25 mm), strong proteins that connect nearly all cells together. Our bodies are full of these, so in a sense, we’re all “one big scar”, but where injuries have healed, they’re unusually densely concentrated, which is why scars are whitish and stiff. Tendons and other strong-stringy tissue also have high collagen concentration, so in a sense are “planned scars”.

 

Scabbing and scarring are neatly organized body repair mechanisms, controlled by a complicated system of chemical signals. Though these mechanism resemble normal growth, they’re really a very different mechanism, involving mostly chemically signaling to specialized cells, rather than genetically programmed protein making from “blueprints” in cells’ DNA.

 

Cancer happens when a cells DNA has a series of malfunctions that make the cell reproduce abnormally, and fail to detect they’re malfunctioning and self-destruct (“suicide”), detect when they have divided many times and self-desctuct, ignore signals from outside to self-destruct, and fight cells sent to kill them. Cancer “hijacks” cells, deactivating their normal safeguards and using their metabolic machinery to make more of themselves.

 

So, unlike scabs, which are cells with very short, well-controlled lifetimes, and scars, which aren’t living cells at all, but protein that connect cells, cancerous cells are “immortal”, “super alive”, reproducing faster and using more nutrients than ordinary cells and, dying of old age, and hard for the body’s natural cell-killer systems, or various artificial medical therapies, to kill. Unlike scabs, cancer cells don’t make barriers against infection, and although specialize invader-fighting cells such as lymphocytes can get cancer, such cancerous cells don’t work better than before, but instead usually use them to spread the cancer.

 

Medically, it’s hard – I think impossible – to find anything beneficial about cancer.

 

In a sense, cancer is the opposite of scabs and scars, with normal cells falling somewhere between these two extremes.

[hardy5]

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